In a lab-grown cell study focused on potential new treatment targets for halting the spread of most pancreatic cancers, Johns Hopkins Medicine scientists report they have found that a gene called KLF5 (Krueppel-like factor 5) fuels the growth of such spreading tumors, not by acquiring abnormal changes in the cancer cells’ DNA code itself, but by altering chemical changes and organization of DNA, or epigenetics, that turns genes on and off.
This article was originally published on MedicalXpress.com
