NMNAT2 is a key protein required for axon integrity whose rapid depletion following axon injury triggers Wallerian degeneration. The molecular mechanism controlling NMNAT2 turnover in neurons has not been fully understood. It has been believed that NMNAT2 degradation is accelerated in axons upon injury, and that NMNAT2 might have different regulatory mechanisms in neuronal soma and axons.
This article was originally published on MedicalXpress.com
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